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Atrioventricular Blocks

1st Degree

Atrioventricular (or AV) heart blocks are usually caused by a delayed, absent, or inconsistent electrical conduction pathway through the AV node.  These AV blocks are described as a first degree, having a prolonged PR interval beyond .20 seconds.

An ECG will show the following:

There is usually a minimal clinical significance with this form of heart block.

2nd Degree Type 1 (Wenckebach)

A second degree, also called a Mobitz Type 1 block, usually has a progressively widening PR interval. This is followed by a QRS complex that is progressively delayed at the AV node until completely absent. It then only shows a P wave without a QRS following it.

An ECG will show the following:

This is commonly caused by a heart disease affecting the AV node or by vagal stimulation, which is often associated with:

2nd Degree Type 2 (Mobitz)

The third type is called Second Degree Mobitz Type 2 and usually occurs when the heart block is below the AV node.

Usually the ECG appears to have intermittent blocks where some P waves do not have QRS complexes following. There is no elongation of the PR interval. In the case of this Type 2 heart block, the rhythm is variable depending on the P to QRS ratio.

An ECG will show the following:

This Type 2 heart block is usually from a more advanced and severe heart disease. It can originate from damage below the bundle of His. Because of this, Mobitz Type 2 can deteriorate more quickly into a symptomatic dysrhythmia and even become a third degree heart block.

3rd Degree Complete Block

Lastly, the third degree complete AV heart block occurs when electrical conduction is completely blocked between the atria and ventricles. The exact location of the block can vary but is usually somewhere around the AV node or lower. It will disassociate the SA pacemaker from the AV or bundle of His pacemakers, creating an ECG with regular P waves and regular QRS waves. But these waves will be at different rates and completely disassociated.

An ECG will show the following:

The clinical significance of this dysrhythmia is serious.  The patient will usually be symptomatic and unstable due to the very slow bradycardic rhythm.

Remember, the block is stopping any pace that would originate from the SA node, so the ventricular pacemaker will stimulate a pulse rate of closer to 20-40 beats per minute. This is usually not enough to keep a stable blood pressure. This is why the ECG will usually have wide QRS complexes.

Studies have shown that third degree heart blocks may be transient or permanent depending on the underlying cause.

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